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发布于:2018-11-9 09:34:04  访问:5 次 回复:0 篇
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Sculitis: <a href="https://www.ncbi.nlm.nih.gov/pubmed/28878015" title
Late post-gadolinium myocardial D a manual sphygmomanometer.Systolic blood pressure (SBP) in the brachial enhancement photos in different planes show septal intramural and anteroseptal and anterior subendocardial lesions. 3Division of Rheumatology, The Ohio State University, 480 Medical Center Drive, S2056 DMRC, Columbus, Oh 43210, USA. Ber of participants. The diagnostic accuracy was 66.7 for <60 years and 56.7 >60 years Received: 15 April 2012 Accepted: 16 November 2012 Published: 30 November 2012 References 1. Scott DG, Watts RA: Systemic vasculitis: epidemiology, classification and environmental aspects.Sculitis: PubMed ID:https://www.ncbi.nlm.nih.gov/pubmed/28878015 Churg-Strauss syndrome (CSS)Patients with tiny vessel vasculitis have important constitutional symptoms. Other a lot more precise complaintsare related for the particular bed that may be affected. One example is, palpable purpura may be the hallmark dermatological manifestation of small vessel vasculitis, whereas alveolar hemorrhage might happen if the lung parenchyma is involved. Clinical clues towards the diagnosis of CSS may perhaps include asthma, hypereosinophilia and fever, and half of these patients test good for anti-neutrophilic cytoplasmic antibody(ANCA) [3]. CSS warrants unique consideration in this assessment given that rates of cardiac involvement happen to be reported to become as higher as 75 to 90 . Cardiovascular involvement in CSS consists of pericarditis, myocarditis, cardiomyopathies and intracavitary thrombi [70,71].In a series of 11 patients undergoing CMR, various forms of myocardial injury was detected in all patients. Imply LVEF was 45 with impairment of LV function in six patients, edema in 4, pericardial effusion in 7, and LGE-positivity in 9 like some with standard LV size and EF [72]. The subendocardial pattern of myocardial involvement in CSS can beFigure 12 A 25 year-old Middle Eastern male with a 6 year history of recurrent deep venous thrombi despite therapeutic anticoagulation presented to a vascular medicine specialist. Physical examination demonstrated painful erythematous lesions more than the lateral aspect in the calf (A, Courtesy Dr. Steven Dean) and oral ulcerations. B. Subsequent contrast-enhanced magnetic resonance venogram showed occlusion of a previously placed filter inside the inferior vena cava (arrowhead). Substantial venous collaterals are evident. C. Coronal plane postcontrast volumetric interpolated breathhold T1-weighted image shows signal void delineating the IVC filter (arrows).Raman et al. Journal of Cardiovascular Magnetic Resonance 2012, 14:82 http://www.jcmr-online.com/content/14/1/Page 12 ofFigure 13 A 37-year-old female with biopsy-proven Churg-Strauss-vasculitis was referred for CMR examination The left ventricle was slightly enlarged with mild systolic dysfunction: LV ejection fraction was 45 . Late post-gadolinium myocardial enhancement pictures in numerous planes show septal intramural and anteroseptal and anterior subendocardial lesions. Photos courtesy Drs. Ralf Wamuth and Jeanette Schulz-Menger.readily detected by LGE-CMR but not echocardiography [73,74]. Existing data help the use of LGE and T2 myocardial imaging in defining the presence and extent of cardiac disease (Figure 13) [70,72]. Future studies using perfusion CMR may perhaps assistance elucidate to what extent microvascular disease contributes to myocardial involvement.Authors‘ contributions SVR drafted the manuscript and figures. WNJ and AA contributed to manuscript preparation and revision. All authors study and authorized the final manuscript. Acknowledgements The authors thank Tam Tran for his assistance in literature compilation. Author information 1 The Ohio State University, 473 W. 12th Ave, Suite 200, Columbus, OH 43210, USA.
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